Extruded complete dietetic food for adult cats to support renal function in CKD..
- High quality Protein and Phosphorus
- Less than 32% protein
- PNP 10
Fish (white fish hydrolysate, fresh Salmon min. 10%), sweet Potato, pulses (Lentils min. 5%), poultry hydrolysate, Potato, Chicken fat, Brewer’s yeast, (Saccharomyces cerevisiae), FAEC Complete (Yucca schidigera extract, yeast products, (Saccharomyces cerevisiae, as a source of MOS and nucleotides), glucosamine, chondroitin sulphate, methylsulfonylmethane, Chicory root, Salvia rosmarinus, Melissa officinalis, Taraxacum officinale, Salvia officinalis, Minthostachys verticillata, Cynara scolymus, Silybum marianum), Carrot, Pumpkin, Apple, whole dehydrated Egg, FAEC Inmune (microalgae oil Schizochytrium sp. (source of Omega 3 DHA fatty acids), Panax ginseng, Punica granatum, Zingiber officinale, Urtica dioica, Camellia sinensis, Curcuma longa, Harpagophytum procumbens), FAEC ProbioDigestive (Enterococcus faecium, Lactobacillus plantarum, Lactobacillus acidophilus, Lactobacillus rhamnosus).
Protein sources: white fish hydrolysate, fresh Salmon, Lentils, poultry hydrolysate, whole dehydrated Egg.
Crude Protein 26%, crude Fat 19%, crude Ash 6.5%, crude Fibre 4.2%, Calcium 0.8%, Phosphorus 0.5%, Potassium 0.8%, Sodium 0.2%, Essential fatty acids 4.85%, DHA+EPA 0.49%, ME 3970 Kcal/kg.
Upports kidney function in case of CKD
Renal disease in cats, as a disease, is very relevant in veterinary practice. Up to 20% of the feline population suffers or will suffer from CKD, a figure that will worsen with age. The various causes of the disease include renal neoplasms or lithiasis, and their effective diagnosis or qualification is of vital importance for their correct treatment or prevention.
Diagnosis will determine the success of treatment, as this is an irreversible disease, with an average life expectancy of 3 years from diagnosis, and the cat’s quality of life will be the main objective. Given that CKD is characterized by structural or functional normality of one or both kidneys, and that the organ itself compensates for this loss with increased glomerular filtration leading to further damage to healthy glomeruli, it is particularly difficult to determine the stage of the disease. By the time azotemia appears and is identified, about 60-65% of functional capacity is usually lost, and the appearance of signs such as polyuria, polydipsia, weight loss, lethargy… are already clear.
It is possible to establish the disease and its evolution through the study of proteinuria, urine density, GFR with creatinine and urea readings, accompanied by analytical tests, diagnostic imaging, etc.
In the nutritional treatment of CKD, we usually opt for protein restriction and subsequent reduction of the load on the kidney, without going overboard in the reduction of protein since the cat, as a facultative carnivore, requires a higher amount of protein than, for example, dogs. Another factor to be taken care of in a renal diet is phosphorus which must be reduced. In cases where this is not possible due to the nature of the recipe, it is interesting to use phosphorus chelators, also in dogs where phosphorus control alone is not sufficient to combat hyperphosphatemia.
If we talk about sodium in the diet, a more restricted level than in a physiological diet is recommended as the ability to excrete sodium is lower in a cat with less functional nephrons, which could lead to high blood pressure. Conversely, hypokalemia is equally common and therefore a potassium adjustment of the diet is recommended. Finally, the use of omega-3 fatty acids limits the appearance of inflammatory mediators, helps to reduce glomerular hypertension and proteinuria, which, combined with ingredients with antioxidant capacity, will help to reduce oxidative damage to the kidneys of CKD patients. Of course, the use of probiotics, especially Lactobacillus acidophilus and Lactobacillus plantarum, have been reported to be beneficial in humans, canids and felines in the treatment of CKD, either by controlling oxidative damage, restoration of the intestinal microbiota, lowering of BUN and, still under study, multiple other benefits in both treatment and prevention are suggested. prevention.